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Introduction
The patient was admitted with hypotension, hypoxia, and ventilator dependent respiratory failure. Upon initial examination, septic shock was assumed as the diagnosis, which was subsequently confirmed through the failure to maintain the patients blood pressure by intravenous fluid administration, which is a sign of resistance to hypotension treatment characteristic of septic shock (Yealy et al., 2014). Most dangerous possible complications include multiple organ dysfunction (heart, kidney, respiratory, liver).
Important aspects of the patients past medical history are chronic respiratory failure, a cerebrovascular accident with subsequent left hemiplegia, multidrug-resistant with recurrent infections, and epilepsy. The patients advance directive allows for all interventions that may be required to restore breathing or heart functioning in case of failures; these possible interventions effectively include the application of a breathing tube and defibrillation.
The patient has spent two days at the hospital. The patient lives with family and receives support from them as well as a visiting social support person. Transferring the patient to a nursing home has been repeatedly discussed. Recommendations proposed by physicians are in favor of this option. The patient was previously treated with gastrostomy and feeding tube placement, which determines certain potential discharge needs, such as constant monitoring for alerts. The patients bedbound status is debatable: a patient can transfer to a chair and sit up.
Pathophysiology
The patient has multiple medical conditions, which shapes the complexity of the clinical picture and patient experiences. It is necessary to take into consideration various aspects of the pathophysiological developments to address the complex state effectively. The most pertinent diagnoses include septic shock, anemia with tachycardia, and toxic metabolic encephalopathy. The pathophysiology of septic shock is understood through severe sepsis processes associated with the immune systems and blood coagulation responses to infection normally caused by bacteria (Yealy et al., 2014). In response to inflammation caused by infection, anti-inflammatory substances are produced by the patients organism, which constitutes the compensatory anti-inflammatory response that leads to suppression of the immune system. Suppressed immune system presents risks of secondary infections.
Anemia with tachycardia is a state of increased cardiac output as a response to the low concentration of hemoglobin (Camaschella, 2015). Toxic metabolic encephalopathy occurs when various substances participating in metabolism are altered and begin to negatively affect the brain (Sharma, Eesa, & Scott, 2012). These substances may include water and electrolytes and may come from kidney and liver waste. Other diagnoses of the patient include malnutrition, decubitus ulcer stage four (due to the patients lifestyle; deep wound affecting muscles and possibly bones with the formation of eschar), ischemic colitis (poor blood supply of the large intestine causing damage and inflammation processes), and thrombocytopenia (a low number of platelets in the blood that may cause poor coagulation and various associated complications, such as internal bleedings).
Medication Regimen
Table 1. All Scheduled Medications and All PRN Medications Given within the Last 48 Hours.
Considering the number of applied medications and the vastness of health problems addressed, the drug interactions should be examined with scrutiny. The patients age (55 years old) is an age of risk for several medications listed above. Also, the patients history of heart problems should be taken into account with possible reduction of the use of those drugs that indicate fast heart rate as their side effects. Also, a particularly challenging drug interaction in this case is the use of levetiracetam and meropenem. Among many antibacterials, meropenem is known to possibly cause seizures (Tanaka et al., 2013), which is why its use in this case may need to be reconsidered.
Laboratory and Diagnostic Data
Table 2. Laboratory Tests Results and Comments.
Assessment
Physical assessment of the patient detected such characteristics and developments as generalized edema, diminished lung sounds, and hypoactive bowel sounds, which are abnormal and serve as possible indicators of inflammation, sepsis, and septic shock. Additional indicators needed for confirming the diagnosis come from assessing microvascular and cellular changes. Activation of inflammatory and coagulation cascades can be observed along with the dilation of blood vessels and the dysfunctional distribution of microvascular blood flow. Combined with the laboratory test results, physical assessment may indicate maldistribution of oxygen and nutrients on the microvascular level. All these developments may not be clearly displayed in vital signs, which makes them hard to detect under the conditions of a clinical examination.
However, vital signs examination can indicate certain relevant developments, such as tachycardia (fast heart rate, more than 100 BPM) and fever (presenting evidence for inflammation processes). Also, oxygen / ventilation settings were reduced, which showed the necessity for appropriate drugs (see Medication Regimen), and the patient had an indwelling catheter.
Cultural and Spiritual Aspects of Care
It has been repeatedly stressed by researchers and health care practitioners that cultural differences and religious or ethnical characteristics of patients can become barriers in providing high-quality care (Levesque, Harris, & Russell, 2013). The fundamental reason for that is a lack of understanding between medical staff and a patient. Also, there are often cases where a patient may be unwilling to follow treatment instructions or cooperate with physicians and nurses due to his or her beliefs. In this context, traditions should not be disregarded, as the communication between patients and providers, which is a prerequisite for appropriate care, can be undermined if a nurse or a doctor fails to adopt such forms of communication and interaction that are acceptable and appropriate according to the patients cultural norms.
In the examined case, no such complications are expected because the patient and the medical staff share cultural context. The members of the medical team are diverse, i.e. they are representatives of different ethnicities and religions, but years of working together have taught them to overcome possible misunderstandings and cooperate effectively. This indicates that health care providers will also be able to find appropriate forms of communicating with the patient.
Another important aspect of care is family engagement. For better patient outcomes, the medical team should strive for establishing favorable relationships with the patients family members and close one. Since the patient is critically ill, there is a need for intensive psychological support from the family, and a role of a nurse in this context is to create an environment where family members understand their role in treatment and their importance for it and are encouraged to provide the patient with necessary support.
Nursing Plan of Care
Table 3. Actual Nursing Diagnoses.
References
Camaschella, C. (2015). Iron-deficiency anemia. New England Journal of Medicine, 372(19), 1832-1843.
Levesque, J. F., Harris, M. F., & Russell, G. (2013). Patient-centred access to health care: conceptualising access at the interface of health systems and populations. International Journal for Equity in Health, 12(1), 18-29.
Sharma, P., Eesa, M., & Scott, J. N. (2012). Toxic and acquired metabolic encephalopathies: MRI appearance. American Journal of Roentgenology, 193(1), 879-886.
Tanaka, A., Takechi, K., Watanabe, S., Tanaka, M., Suemaru, K., & Araki, H. (2013). Comparison of the prevalence of convulsions associated with the use of cefepime and meropenem. International Journal of Clinical Pharmacy, 35(5), 683-687.
Yealy, D. M., Kellum, J. A., Huang, D. T., Barnato, A. E., Weissfeld, L. A., Pike, F., & Angus, D. C. (2014). A randomized trial of protocol-based care for early septic shock. New England Journal of Medicine, 370(1), 1683-1693.
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